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Animal 12 (s1), s133s141

Presented for research literature review only Halder M et al

69 In gastric cancer cells, the absence of CDO1 contributed to restore cellular glutathione levels, to prevent ROS and lipid peroxidation, and to promote resistance to ferroptosis, a type of programmed cell death dependent on iron and associated with lipid peroxide accumulation

Both require identical storagelyophilized at -20C, reconstituted at 2-8C, used within 28 days

Transport of cystine and cysteine in mammalian cells

To explain these results, we investigate FSP1 expression in MDA-MB-231 compared to other BC cell lines since the oxidized form CoQ 10 needs to be reduced by the NADH-dependent oxidoreductases FSP1 to CoQ 10 H 2 , and only CoQ 10 H 2 acts as a radical-trapping antioxidant to suppress ferroptosis 59